MRL-24

• Background:

  Displays high affinity for PPARγ with excellent anti-diabetic activity in mice but poor PPARγ agonist activity in transcription and adipogenesis assays. Highly effective (30 nM) at blocking Cdk5-mediated phosphorylation of PPARγ. Cell permeable.

• Conjugation:

  Unconjugated

• Buffer:

  White solid

• Storage Conditions:

  RT