Human FXR1 CRISPR Knock Out 293T Cell Line

Cell line type

293T
NCBI number

NM_001013438
Availability

Please contact us every day to check the availability and/or to request a quote. For larger or bulk quantities a discount may apply.
Ordering

If you wish to order this product, please include the catalogue number #K0823751in your purchase order among with your shipping and billing information.
Technical file

Please contact us to request the latest datasheet, protocol, certificate of analysis and SDS files.
Notes

Designed for Research Use Only. Manufactured in Canada.
Other products
Description

For cells, cell lines and tissues in culture till half confluency.
Properties

Human proteins, cDNA and human recombinants are used in human reactive ELISA kits and to produce anti-human mono and polyclonal antibodies. Modern humans (Homo sapiens, primarily ssp. Homo sapiens sapiens). Depending on the epitopes used human ELISA kits can be cross reactive to many other species. Mainly analyzed are human serum, plasma, urine, saliva, human cell culture supernatants and biological samples.

Gene target

FXR1 CRISPR Knock Out Line
Gene symbol

FXR1
Short name

FXR1 CRISPR Knock Out Line
Technique

CV40 transformed cell line or 293tsA1609neo from human kidney 293 cells delivered as 1.
Species

Human, Humans
Alternative name

H. sapiens fragile X mental retardation, autosomal homolog 1 CRISPR Knock Out 293T cellular Line
Alternative to gene target

fragile X mental retardation, autosomal homolog 1, FXR1P, FXR1 and IDBG-66609 and ENSG00000114416 and 8087, poly(A) RNA binding, nuclei, Fxr1 and IDBG-137735 and ENSMUSG00000027680 and 14359, FXR1 and IDBG-635881 and ENSBTAG00000016457 and 536793
Tissue

cell

Name

Transformation, Bacterial
Concept

Scope note: The heritable modification of the properties of a competent bacterium by naked DNA from another source. The uptake of naked DNA is a naturally occuring phenomenon in some bacteria. It is often used as a GENE TRANSFER TECHNIQUE.
Tree numbers
- E05.393.350.810.500
Qualifiers

immunology, physiology, ethics, drug effects, radiation effects, genetics